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by Marijana Domazet, Wednesday, 29 May 2013 | Categories: Obesity

It should come as no surprise that the obesity epidemic is not restricted to the adult population. Whilst reports on adult obesity have tended to focus on endocrinology, inappropriate diets and obstacles to exercise, reports on childhood obesity have sought to explore whether socioeconomic status and neurological processes could account for the development of obesity. Here we consider the most recent study in this field, which explored the potential relationship between body weight and ADHD.

The study, which was published in PlosOne, assessed 284 children with ADHD between the ages of six to twelve. It came as an extension of a previous study, which had conducted less thorough neurocognitive assessments of children with ADHD. The initial study, which was not done by the same team, had concluded that both ADHD and obesity could be due to impaired self-regulatory processes. However, as this study had used crude measures on a limited sample, and even tested the children who were currently taking ADHD medications, there was limited support for the findings.

In an attempt to address all of those limitations, the researchers of the current study ensured that all the participants had not taken their medications for at least a week before taking part. They then assessed them using a battery of tests, which included a more realistic reflection of typical neurocognitive characteristics, motivational styles and motor profiles that could be useful to explain the findings. In addition to that, the data was adjusted for demographic characteristics, so as to account for environmental influences. The key findings indicated that there was little evidence for the relationship between neurocognitive deficits and obesity, but that socioeconomic status was a useful predictor for obesity. This led the researchers to argue that the effects of socioeconomic status, such as not having access to exercise or a regular healthy diet, were better predictors than self-regulatory processes.

It is not hard to understand the rationale behind this study as it comes within a context of reports of patients with eating disorders having attention difficulties that were analogous to ADHD. Clinically, there have also been some neuroimaging studies that have suggested that patients with ADHD exhibit the dopamine dysregulation that can be seen in individuals with food addiction.

However, it is worth mentioning that the food addiction model is questionable at this stage, and that the comparison between clinical disorders is far too superficial. ADHD, obesity and eating disorders are complex multifaceted phenomena that are not yet fully understood. Consequently, for this line of reasoning to be considered, more extensive large-scale studies need to be put in place.

Having said that, there are still aspects of the current study that should not be ignored. Understanding how lower socioeconomic status affects the development of obesity is crucial as there may be a way to address these risk factors socially. Therefore, this study may be a cause for optimism in the battle against childhood obesity.





 
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